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Monday, October 24, 2011 — Concurrent Symposia Session I | |
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2:00 p.m. – 4:00 p.m. |
Balcony B |
A cross-section of NIH and USUHS scientists who are participating in both formal and informal collaborations to decipher the mechanisms of mild traumatic brain injury underlying post-traumatic stress disorder (PTSD) and other long-term cognitive complications of mild traumatic brain injury (mTBI) will explore this issue from several distinct points of view: pre-conditioning of brain responses to ischemia and insult; long-term effects on hippocampal function of non-penetrating injury; neuroprotection by endogenous brain transmitters and synthetic ligands, including epigenetic mechanisms for sparing of brain function; and relationships between clinical emotional disorders and sub-clinical organic brain injury. "Unsiloing" these distinct but critically interdependent aspects of mTBI and PTSD should be of intense interest to the translational community, both military- and civilian-oriented, on the NIH and USUHS campuses.
Introduction to Protecting the Brain from Traumatic InjuryPTSD and Traumatic Brain Injury: Defining the Problem
Robert Ursano, USUHS
Boosting Endogenous Stress Resistance in the Brain to Counter the Consequences of CNS Insults
John Hallenbeck, NINDS
Stroke and Traumatic Brain Injury: Common Mechanisms and Potential Treatments
De-Maw Chuang, NIMH
Alterations in Hippocampal Function and Gene Expression After Controlled Cortical Injury
Maria Braga, USUHS
Boosting Endogenous Cannabinoids to Alleviate the Effects of Psychological Trauma
Andrew Holmes, NIAAA
Analysis of Brain Network Dynamics after Transcranial Direct Current Stimulation
*FARE Award Winner
Anusha Venkatakrishnan, NINDS