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Home > Concurent Symposium Sessions > Role of TOR in Cell Growth and Cancer

Concurent Symposium Sessions
  Tuesday, September 25
Natcher Conference Center
Symposia Session I

Role of TOR in Cell Growth and Cancer

2:00 pm to 4:00 pm

Co-Chaired by:
Beverly Mock, NCI/CCR and Alan Kimmel, NIDDK

Natcher Conference Center - Balcony A

The target of rapamycin (TOR) is central to cell growth and nutrient sensing pathways. The ability of TOR to phosphorylate substrates such as p70S6 kinase, 4E-BP1, Akt and p53, place it as a key signaling node involved in many pathways which have the potential to modulate both developmental and disease-related processes as diverse as stem cell fate, diabetes, aging and cancer. This symposium will highlight the TOR research of NIH investigators from different institutes and disciplines.


The Antagonistic Actions of TOR Complexes 1 & 2
Alan Kimmel, NIDDK

TOR Complex 2 Integrates Chemotaxis and Signal Relay in Dictyostelium
Carole Parent, NCI/CCR

mTOR Activity Regulates Stem Cell Fate
Toren Finkel, NHLBI

The Role of mTOR in Pediatric Cancers
Lee Helman, NCI/CCR

mTOR acts as a Tumor Suppressor in Plasma Cell Tumor Development
Beverly Mock, NCI/CCR

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